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They studied postmortem liver tissue of schizophrenia patients and found an upregulation of SOD2, 3-phosphoinositide-dependent protein kinase (PDK1) , and NSF Attachment Protein Gamma (NAPG) and downregulation of 11 mitochondria-related genes, including D-beta-hydroxybutyrate dehydrogenase (BDH1), acyl-coenzyme A dehydrogenase (ACAMD), Solute carrier family 25 member 47 (SLC25A47) , and N-Acetylglutamate synthase (NAGS) in patients treated with typical antipsychotic compared to atypical antipsychotic treatment [32]
