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Previous studies have shown that SIRT1 enhances PGC-1α expression and promotes mitochondrial biogenesis, with PGC-1α knockdown nearly abolishing SIRT1’s neuroprotective effect in SH-SY5Y cells 41
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In an inflammatory state, knockout of Clec16a in β cells can significantly increase apoptosis and sharply increase blood glucose levels in patients, and hyperglycemia further affects mitochondrial quality control, promotes cell damage, and exacerbates diabetes
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![PDF] Oral Administration of S-acetyl-glutathione: Impact on the Levels of Glutathione in Plasma and in Erythrocytes of Healthy Volunteers](https://figures.semanticscholar.org/fb2b6ad5ffd26d47b18fdf9324779b69da49dbcc/5-Table2-1.png)
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