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The CAL liberated by IFO in particular can be further metabolized by adduct formation with cellular thiols leading to S -carboxymethyl-L-cysteine (SCMC) and the depletion of total cysteine, glutathione and homocysteine (Lauterburg et al., 1994)
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Thus, regulation of ferroptosis by TCM may become a means of treating NAFLD (Table 1)
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