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There is evidence that in these regions, where individuals are repeatedly exposed to infection, there is development of more long-lived antibodies compared to areas with low transmission (65)
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This review summarizes current evidence regarding the role of ferroptosis in osteoporosis, with particular focus on its effects on osteoblasts, osteoclasts, and osteocytes, as well as the impact of aging, estrogen deficiency, diabetes, glucocorticoid exposure, and obesity on ferroptosis in osteogenic cells
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