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Thymosin Alpha-1 drives muscularis macrophage polarisation from M1 (ICC/nNOS-injurious) to M2 (ICC-supportive, BMP2/CSF1-secreting), achieving the strongest ICC density research applications across compounds studied

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Oxidative mechanisms may also regulate GPx-1 gene transcription via nuclear factor κB (NFκB) sites and activator protein 1 (AP-1) sites in the promoter