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This disrupts the balance between Th 17 and Treg cells, and the resulting proinflammatory microenvironment, dominated by Th 1 and Th 17 cells, further exacerbates the damage to thyroid tissue

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This three-step process involves the conversion of dietary Met—primarily found in eggs, meat, and legumes—into S-adenosyl Met (SAM), then into S-adenosyl Hcy (SAH) and, finally, into Hcy [1]